| 公開日期 | 標題 | 作者 | 來源出版物 | scopus | WOS | 全文 |
1 | 2019 | The antimicrobial photodynamic inactivation resistance of Candida albicans is modulated by the Hog1 pathway and the Cap1 transcription factor | Chien, C.-T.; Chen, Y.-C.; Liu, Y.-C.; Liang, S.-H.; Lin, H.-H.; CHING-HSUAN LIN | Medical mycology | 12 | 13 | |
2 | 2019 | A Potential Antifungal Effect of Chitosan Against Candida albicans Is Mediated via the Inhibition of SAGA Complex Component Expression and the Subsequent Alteration of Cell Surface Integrity | P. Y. Shih; Y. T. Liao; Y. K. Tseng; F. S. Deng; C. H. Lin | Frontiers in Microbiology | 71 | 56 | |
3 | 2018 | Chitosan Inhibits the Rehabilitation of Damaged Microbes Induced by Photodynamic Inactivation | Lin, Ching-Hsuan; Chien, Hsiung-Fei; Lin, Ming-Hsuan; Chen, Chueh-Pin; Shen, Mandy; C. T. Chen ; C. H. Lin | International journal of molecular sciences | 12 | 10 | |
4 | 2018 | Cpp1 phosphatase mediated signaling crosstalk between Hog1 and Cek1 mitogen activated protein kinases is involved in the phenotypic transition in Candida albicans | F. S. Deng; CHING-HSUAN LIN | Medical Mycology | 13 | 11 | |
5 | 2018 | Identification and characterization of ORF19.1725, a novel gene contributing to the white cell pheromone response and virulence-associated functions in Candida albicans | Deng, Fu-Sheng; CHING-HSUAN LIN | Virulence | 5 | 5 | |
6 | 2016 | The conserved dual phosphorylation sites of the Candida albicans Hog1 protein are crucial for white-opaque switching, mating, and pheromone-stimulated cell adhesion. | Wen-Han Chang; Shen-Huan Liang; Fu-Sheng Deng; Ching-Hsuan Lin; CHING-HSUAN LIN | Medical mycology | 12 | 10 | |
7 | 2015 | Correction for Liang et al., A Novel Function for Hog1 Stress-Activated Protein Kinase in Controlling White-Opaque Switching and Mating in Candida albicans | Liang, Shen-Huan; Cheng, Jen-Hua; Deng, Fu-Sheng; Tsai, Pei-An; Lin, Ching-Hsuan; CHING-HSUAN LIN | Eukaryotic Cell | 0 | 0 | |
8 | 2014 | Interactions of MAP Kinases, Histidine Kinase and YAP1 in the Citrus Fungal Pathogen Alternaria alternata | CHING-HSUAN LIN | Plant Pathology bulletin | | | |
9 | 2014 | A novel function for Hog1 Stress-Activated protein kinase in controlling white-opaque switching and mating in candida albicans | Liang, S.-H.; Cheng, J.-H.; Deng, F.-S.; Tsai, P.-A.; Lin, C.-H.; CHING-HSUAN LIN | Eukaryotic Cell | 30 | 26 | |
10 | 2013 | Genetic Control of Conventional and Pheromone-Stimulated Biofilm Formation in Candida albicans | Lin, C.-H.; Kabrawala, S.; Fox, E.P.; Nobile, C.J.; Johnson, A.D.; Bennett, R.J.; CHING-HSUAN LIN | PLOS Pathogens | 78 | 71 | |
11 | 2013 | A nonribosomal peptide synthetase mediates siderophore production and virulence in the citrus fungal pathogen Alternaria alternata | Chen, L.-H.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Molecular Plant Pathology | 66 | 65 | |
12 | 2012 | Roles for SKN7 response regulator in stress resistance, conidiation and virulence in the citrus pathogen Alternaria alternata | Chen, L.-H.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Fungal Genetics and Biology | 49 | 47 | |
13 | 2011 | Gene inactivation in the citrus pathogenic fungus Alternaria alternata defect at the Ku70 locus associated with non-homologous end joining | Wang, N.-Y.; Yang, S.L.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | World Journal of Microbiology and Biotechnology | 14 | 13 | |
14 | 2011 | Defining pheromone-receptor signaling in Candida albicans and related asexual Candida species | Lin, C.-H.; Choi, A.; Bennett, R.J.; CHING-HSUAN LIN | Molecular Biology of the Cell | 17 | 14 | |
15 | 2011 | The SLT2 mitogen-activated protein kinase-mediated signalling pathway governs conidiation, morphogenesis, fungal virulence and production of toxin and melanin in the tangerine pathotype of Alternaria alternata | Yago, J.I.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Molecular Plant Pathology | 60 | 59 | |
16 | 2011 | Cellular responses required for oxidative stress tolerance, colonization, and lesion formation by the necrotrophic fungus Alternaria alternata in Citrus | Lin, C.-H.; Yang, S.L.; Chung, K.-R.; CHING-HSUAN LIN | Current Microbiology | 41 | 40 | |
17 | 2010 | Characterization of Xanthomonas campestris pv. campestris heat shock protein A (HspA), which possesses an intrinsic ability to reactivate inactivated proteins | Lin, C.-H.; Lee, C.-N.; Lin, J.-W.; Tsai, W.-J.; Wang, S.-W.; Weng, S.-F.; Tseng, Y.-H.; CHING-HSUAN LIN | Applied Microbiology and Biotechnology | 12 | 9 | |
18 | 2010 | A Gα subunit gene is essential for conidiation and potassium efflux but dispensable for pathogenicity of Alternaria alternata on citrus | Wang, N.-Y.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Current Genetics | 22 | 22 | |
19 | 2010 | The FUS3 MAPK signaling pathway of the citrus pathogen Alternaria alternata functions independently or cooperatively with the fungal redox-responsive AP1 regulator for diverse developmental, physiological and pathogenic processes | Lin, C.-H.; Yang, S.L.; Wang, N.-Y.; Chung, K.-R.; CHING-HSUAN LIN | Fungal Genetics and Biology | 44 | 42 | |
20 | 2010 | Specialized and shared functions of the histidine kinase- and HOG1 MAP kinase-mediated signaling pathways in Alternaria alternata, a filamentous fungal pathogen of citrus | Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Fungal Genetics and Biology | 81 | 79 | |
21 | 2009 | Coordinate control of oxidative stress tolerance, vegetative growth, and fungal pathogenicity via the AP1 pathway in the rough lemon pathotype of Alternaria alternata | Yang, S.L.; Lin, C.-H.; Chung, K.-R.; CHING-HSUAN LIN | Physiological and Molecular Plant Pathology | 37 | 35 | |
22 | 2009 | The YAP1 homolog-mediated oxidative stress tolerance is crucial for pathogenicity of the necrotrophic fungus alternaria alternata in citrus | Lin, C.-H.; Yang, S.L.; Chung, K.-R.; CHING-HSUAN LIN | Molecular Plant-Microbe Interactions | 111 | 105 | |
23 | 2007 | Characterization of a novel T4-type Stenotrophomonas maltophilia virulent phage Smp14 | Chen, C.-R.; Lin, C.-H.; Lin, J.-W.; Chang, C.-I.; Tseng, Y.-H.; Weng, S.-F.; CHING-HSUAN LIN | Archives of Microbiology | 26 | 26 | |