The Effects of Short-Term Air Pollution Exposure on Blood Pressure Changes in Nonsmoking Adults
Resource
EPIDEMIOLOGY v.22 n.1 SUPPL. S pp.S159-S159
Journal
Epidemiology
Pages
S159
Date Issued
2011
Date
2011
Author(s)
CHEN, SZU-YING
LIN, YU-LUN
CHAN, CHANG-CHUAN
Abstract
O-31C3-2 Background/Aims: Previous studies demonstrated inconclusive associations between short-term air pollution exposure and blood pressure changes. Methods: We used a population-based approach, including 9238 nonsmokers over 20 years of age, to investigate short-term air pollution exposure on blood pressure changes. Individuals' exposures to sulfur dioxide (SO2), carbon monoxide (CO), ozone (O3), particulate matters with diameter <10 mm (PM10), and nitrogen dioxide (NO2) were calculated from the data of Taiwan Air Quality Monitoring Network. A generalized linear model was applied to analyze the data. Results: For an interquartile range (IQR) of SO2, CO, O3, PM10, and NO2, pulse pressure narrowed 1.0 (95% confidence interval [CI]: −1.2 to −0.7), 1.1 (95% CI: −1.4 to −0.8), 0.6 (95% CI: −0.9 to −0.2), 1.5 (95% CI: −1.9 to −1.1), and 3.0 (95% CI: −3.5 to −2.5) mm Hg; systolic blood pressure decreased 0.6 (95% CI: −1.0 to −0.3), 1.2 (95% CI: −1.4 to −0.9), 0.4 (95% CI: −0.8 to −0.1), 0.5 (95% CI: −0.9 to 0.0), and 2.6 (95% CI: −3.1to −2.0) mm Hg. An IQR of SO2, O3, PM10, and NO2 increased 0.9 (95% CI: 0.8 to 1.1), 0.5 (95% CI: 0.2 to 0.8), 0.6 (95% CI: 0.2 to 0.9), and 1.1 (95% CI: 0.7 to 1.5) mm Hg of diastolic blood pressure. The effects of CO and O3 on narrowing pulse pressure were more significant in female than in male. Conclusion: Short-term air pollution exposure can narrow the pulse pressure, indicating decreased cardiac output. Female are more vulnerable to short-term air pollution exposure.