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  3. Epidemiology and Preventive Medicine / 流行病學與預防醫學研究所
  4. The role of common genetic variation in presumed monogenic epilepsies
 
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The role of common genetic variation in presumed monogenic epilepsies

Journal
EBioMedicine
Journal Volume
81
Date Issued
2022-07
Author(s)
Campbell, Ciarán
Leu, Costin
YEN-CHEN ANNE FENG  
Wolking, Stefan
Moreau, Claudia
Ellis, Colin
Ganesan, Shiva
Martins, Helena
Oliver, Karen
Boothman, Isabelle
Benson, Katherine
Molloy, Anne
Brody, Lawrence
Michaud, Jacques L
Hamdan, Fadi F
Minassian, Berge A
Lerche, Holger
Scheffer, Ingrid E
Sisodiya, Sanjay
Girard, Simon
Cosette, Patrick
Delanty, Norman
Lal, Dennis
Cavalleri, Gianpiero L
DOI
10.1016/j.ebiom.2022.104098
URI
https://scholars.lib.ntu.edu.tw/handle/123456789/639997
URL
https://api.elsevier.com/content/abstract/scopus_id/85131451531
https://www.scopus.com/inward/record.uri?eid=2-s2.0-85131451531&doi=10.1016%2fj.ebiom.2022.104098&partnerID=40&md5=2eb6795807b073b136c0e89c44211c30
Abstract
The developmental and epileptic encephalopathies (DEEs) are the most severe group of epilepsies which co-present with developmental delay and intellectual disability (ID). DEEs usually occur in people without a family history of epilepsy and have emerged as primarily monogenic, with damaging rare mutations found in 50% of patients. Little is known about the genetic architecture of patients with DEEs in whom no pathogenic variant is identified. Polygenic risk scoring (PRS) is a method that measures a person's common genetic burden for a trait or condition. Here, we used PRS to test whether genetic burden for epilepsy is relevant in individuals with DEEs, and other forms of epilepsy with ID.
Subjects
DEEs; Epilepsy; Genetic diagnostics; PRS
Type
journal article

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