GWO-TSANN CHUANGLin, Shih-HuaShih-HuaLinTsau, Yong-KweiYong-KweiTsauI-JUNG TSAI2021-01-132021-01-132016-050929-6646https://scholars.lib.ntu.edu.tw/handle/123456789/540540en[SDGs]SDG3potassium; potassium chloride; spironolactone; SLC12A1 protein, human; sodium potassium chloride cotransporter 2; Bartter syndrome; case report; child; gene; gene mutation; genetic analysis; human; hypercalciuria; hypokalemia; kidney calcification; Letter; male; metabolic alkalosis; nephrogenic diabetes insipidus; polydipsia; polyuria; potassium blood level; prenatal period; preschool child; sodium potassium chloride cotransporter gene; ultrasound; Bartter syndrome; complication; Diabetes Insipidus, Nephrogenic; echography; genetics; mutation; Bartter Syndrome; Child, Preschool; Diabetes Insipidus, Nephrogenic; Humans; Male; Mutation; Solute Carrier Family 12, Member 1; Ultrasonographyjournal article10.1016/j.jfma.2015.03.01725935500WOS:000375040500015