In Vivo CD3+CD25+ Lymphocyte Subpopulation Is Down‐regulated Without Increased Serum‐Soluble Interleukin‐2 Receptor (sIL‐2R) by Gonadotropin Releasing Hormone Agonist (GnRH‐a)

HONG-NERNG HOMING-YIH WUHSIN-FU CHENKUANG-HAN CHAOYU-SHIH YANGHuang, S CS CHuangLee, T YT YLeeGill, T JT JGill2022-11-082022-11-081995-011046-7408https://scholars.lib.ntu.edu.tw/handle/123456789/624497To test further whether the suppression of the CD3+CD25+ lymphocyte subpopulation by gonadotropin-releasing hormone agonist (GnRH-a) is related to the change in levels of cytokines and soluble interleukin-2 receptor (sIL-2R).enCD25 | CD69 | cytokine | Gonadotropin‐releasing hormone | soluble interleukin‐2 receptorIn Vivo CD3+CD25+ Lymphocyte Subpopulation Is Down‐regulated Without Increased Serum‐Soluble Interleukin‐2 Receptor (sIL‐2R) by Gonadotropin Releasing Hormone Agonist (GnRH‐a)journal article10.1111/j.1600-0897.1995.tb01150.x76192282-s2.0-0028925840https://scholars.lib.ntu.edu.tw/handle/123456789/459888