Huang, Hung-LingHung-LingHuangLuo, Yong-ChunYong-ChunLuoLu, Po-LiangPo-LiangLuHuang, Cheng-HsiehCheng-HsiehHuangLin, Kun-DerKun-DerLinMENG-RUI LEECheng, Meng-HsuanMeng-HsuanChengYeh, Yao-TsungYao-TsungYehKao, Cheng-YuanCheng-YuanKaoJANN-YUAN WANGYang, Jinn-MoonJinn-MoonYangChong, Inn-WenInn-WenChong2023-02-152023-02-152023-01-1114659921https://scholars.lib.ntu.edu.tw/handle/123456789/628122Diabetes mellitus (DM) is a major risk factor for tuberculosis (TB). Evidence has linked the DM-related dysbiosis of gut microbiota to modifiable host immunity to Mycobacterium tuberculosis infection. However, the crosslinks between gut microbiota composition and immunological effects on the development of latent TB infection (LTBI) in DM patients remain uncertain.enDiabetic mellitus; Gut microbiota; Immunity; Latent tuberculosis infection[SDGs]SDG3Gut microbiota composition can reflect immune responses of latent tuberculosis infection in patients with poorly controlled diabetesjournal article10.1186/s12931-023-02312-w366318572-s2.0-85146133426https://api.elsevier.com/content/abstract/scopus_id/85146133426