LEE-MING CHUANG2020-06-012020-06-0120102040-1116https://www.scopus.com/inward/record.uri?eid=2-s2.0-79953737536&doi=10.1111%2fj.2040-1124.2010.00042.x&partnerID=40&md5=dc018a035dc34db4f4d2af0bf782023ahttps://scholars.lib.ntu.edu.tw/handle/123456789/495701[SDGs]SDG3glucose; insulin; inwardly rectifying potassium channel subunit Kir6.2; peroxisome proliferator activated receptor gamma; potassium channel KCNQ1; protein PTPRD; receptor like protein tyrosine phosphatase; transcription factor 7 like 2; unclassified drug; causal attribution; cell function; clinical feature; editorial; gene expression; gene identification; gene locus; gene sequence; genetic association; genetic linkage; genetic risk; genetic susceptibility; genetic variability; heritability; human; insulin release; insulin resistance; non insulin dependent diabetes mellitus; pancreas islet beta cell; pathophysiology; phenotype; priority journal; race difference; sequence analysis; single nucleotide polymorphismeditorial10.1111/j.2040-1124.2010.00042.x2-s2.0-79953737536