Acquisition of JAK2, PTPN11, and RAS mutations during disease progression in primary myelodysplastic syndrome [1]

Chen, Chien-YuanChien-YuanChenLIANG-IN LINJIH-LUH TANGWOEI TSAYHSIU-HAO CHANGYeh Y.-C.Huang C.-F.Chiou R.-J.MING YAOBOR-SHENG KOYAO-CHANG CHENLin K.-H.Lin D.-T.HWEI-FANG TIEN2020-06-162020-06-1620060887-6924https://scholars.lib.ntu.edu.tw/handle/123456789/502717[SDGs]SDG3Janus kinase 2; protein tyrosine phosphatase SHP 2; Ras protein; adolescent; adult; aged; allogeneic hematopoietic stem cell transplantation; carcinogenesis; child; chronic myelomonocytic leukemia; codon; controlled study; correlation function; disease course; exon; female; follow up; gene mutation; gene sequence; human; infant; juvenile myelomonocytic leukemia; letter; major clinical study; male; malignant transformation; myelodysplastic syndrome; polymerase chain reaction; priority journal; refractory anemia with excess blasts in transformation; refractory anemia with ringed sideroblasts; statistical significance; thrombocyte countAcquisition of JAK2, PTPN11, and RAS mutations during disease progression in primary myelodysplastic syndrome [1]letter10.1038/sj.leu.2404190165983122-s2.0-33744496772