Liao W.-T.CHIA-LI YULan C.-C.E.Lee C.-H.Chang C.-H.Chang L.W.You H.-L.Yu H.-S.2021-01-272021-01-2720090143-3334https://www.scopus.com/inward/record.uri?eid=2-s2.0-66749117248&doi=10.1093%2fcarcin%2fbgp095&partnerID=40&md5=ccb96bf0df23dee6fb3a0a010e67433ahttps://scholars.lib.ntu.edu.tw/handle/123456789/542200[SDGs]SDG3arsenic; Fas ligand; tumor necrosis factor receptor 1; adult; aged; apoptosis; article; Bowen disease; CD4+ T lymphocyte; chemical carcinogenesis; clinical article; controlled study; female; gene expression; human; human cell; human tissue; immune deficiency; immunofluorescence; keratinocyte; lymphocyte; lymphocytic infiltration; male; mononuclear cell; peripheral blood mononuclear cell; priority journal; skin disease; systemic disease; tumor immunity; Aged; Apoptosis; Arsenic; Bowen's Disease; CD4-Positive T-Lymphocytes; Cell Transformation, Neoplastic; Cells, Cultured; Fas Ligand Protein; Female; Humans; Immunity, Active; Keratinocytes; Male; Middle Aged; Skin NeoplasmsDifferential effects of arsenic on cutaneous and systemic immunity: Focusing on CD4+ cell apoptosis in patients with arsenic-induced Bowen's diseasejournal article10.1093/carcin/bgp095193768472-s2.0-66749117248