Payne S.J.Morris H.J.Rowley A.B.STEPHEN JOHN PAYNE2022-05-242022-05-242005https://www.scopus.com/inward/record.uri?eid=2-s2.0-33846896236&doi=10.1109%2fiembs.2005.1616923&partnerID=40&md5=39771bdfa7b4e4500fae2ca89e01b5b3https://scholars.lib.ntu.edu.tw/handle/123456789/611874The feedback processes that control the blood supply to the brain, collectively termed cerebral autoregulation, are many and complex. Failure of this mechanism is implicated in a number of conditions. However, in a clinical setting the number of variables that can be measured non-invasively is extremely limited. In addition, current models are either too crude, and not physiologically meaningful, or so complex that analysis is difficult. In this paper a new compact, but physiologically meaningful, model is presented, based on smooth muscle cells within a vessel model, integrated within a full haemodynamic model. Preliminary results show realistic behaviour. ? 2005 IEEE.BiochemistryFeedbackHemodynamicsMusclePhysiologyCerebral autoregulationHemodynamic modelsMuscle cellsBrainconference paper10.1109/iembs.2005.16169232-s2.0-33846896236