Wang, Hsin-WeiHsin-WeiWangLi, Hsing-HanHsing-HanLiSHIH-CHENG WUTang, Cheng-KangCheng-KangTangYu, Hui-YingHui-YingYuChang, Ya-ChenYa-ChenChangSung, Pei-ShanPei-ShanSungLiu, Wei-LiangWei-LiangLiuSu, Matthew PMatthew PSuYu, Guann-YiGuann-YiYuHuang, Li-RungLi-RungHuangChen, Chun-HongChun-HongChenHsieh, Shie-LiangShie-LiangHsieh2023-03-062023-03-062023-02-1710217770https://scholars.lib.ntu.edu.tw/handle/123456789/628947Zika virus (ZIKV) infection is clinically known to induce testicular swelling, termed orchitis, and potentially impact male sterility, but the underlying mechanisms remain unclear. Previous reports suggested that C-type lectins play important roles in mediating virus-induced inflammatory reactions and pathogenesis. We thus investigated whether C-type lectins modulate ZIKV-induced testicular damage.enCLEC5A; Inflammation; Mosquito; Mouse; Testicular damage; Zika virusCLEC5A mediates Zika virus-induced testicular damagejournal article10.1186/s12929-023-00906-6368038042-s2.0-85148390083https://api.elsevier.com/content/abstract/scopus_id/85148390083