Wu, Yi-HsiuYi-HsiuWuLai, Alan Chuan-YingAlan Chuan-YingLaiChi, Po-YuPo-YuChiThio, Christina Li-PingChristina Li-PingThioChen, Wei-YuWei-YuChenTsai, Ching-HuiChing-HuiTsaiYUNG-LING LEELukacs, Nicholas WNicholas WLukacsChang, Ya-JenYa-JenChang2020-02-052020-02-052019-10-170105-4538https://scholars.lib.ntu.edu.tw/handle/123456789/453471Respiratory syncytial virus (RSV) infection is epidemiologically linked to asthma. During RSV infection, IL-33 is elevated and promotes immune cell activation, leading to the development of asthma. However, which immune cells are responsible for triggering airway hyperreactivity (AHR), inflammation and eosinophilia remained to be clarified. We aimed to elucidate the individual roles of IL-33-activated innate immune cells, including ILC2s and ST2+ myeloid cells, in RSV infection-triggered pathophysiology.enIL-33; ILC2; asthma; eosinophilia; respiratory syncytial virus[SDGs]SDG3journal article10.1111/all.14091316225072-s2.0-85075030054WOS:000495842300001https://www.scopus.com/inward/record.uri?eid=2-s2.0-85075030054&doi=10.1111%2fall.14091&partnerID=40&md5=feaf1065bf70362aa6594390cafa328c