WEI-CHIH LIAOChen, Peng-RueiPeng-RueiChenHuang, Cheng-ChiehCheng-ChiehHuangChang, Yen-TzuYen-TzuChangHuang, Bo-ShihBo-ShihHuangCHIN-CHEN CHANGMING-SHIANG WULU-PING CHOW2020-11-232020-11-232020-082190-5991https://scholars.lib.ntu.edu.tw/handle/123456789/522060Pancreatic cancer-associated diabetes mellitus (PCDM) is a paraneoplastic phenomenon characterized by worsening hyperglycaemia and weight loss. Galectin-3 and S100A9, mediators of PCDM, have pro-inflammatory functions and might thereby induce systemic inflammation and cachexia. We aimed to examine whether PCDM directly mediates cachexia.enCachexia; Diabetes; Pancreatic cancer; Sarcopenia[SDGs]SDG3calgranulin B; galectin 3; glucose; hemoglobin A1c; adult; Article; body mass; body weight loss; cachexia; cancer size; cancer staging; clinical feature; diabetes mellitus; diabetic patient; disease association; fasting; female; glucose blood level; human; hyperglycemia; logistic regression analysis; major clinical study; male; middle aged; muscle mass; pancreas cancer; paraspinal muscle; primary tumor; priority journal; protein blood level; sarcopenia; skeletal muscle; cachexia; complication; diabetes mellitus; pancreas tumor; pathophysiology; Cachexia; Diabetes Mellitus; Female; Humans; Male; Middle Aged; Pancreatic Neoplasmsjournal article10.1002/jcsm.1255332100478WOS:000517296500001