CHUN-LIANG PANYuki N.Koga M.Chiang M.-C.SUNG-TSANG HSIEH2020-03-022020-03-0220010028-3878https://www.scopus.com/inward/record.uri?eid=2-s2.0-0035833949&doi=10.1212%2fWNL.57.7.1316&partnerID=40&md5=fafd0abb290372abec1d1042083711b3https://scholars.lib.ntu.edu.tw/handle/123456789/467819The authors describe two patients with acute sensory ataxic neuropathy. Both had a profound loss of proprioception and generalized areflexia. High titers of monospecific anti-GD1b IgG antibody were detected in their sera during the acute phase. Sensory ataxia resolved within 2 weeks after the onset. Taken together with the induction of experimental sensory ataxic neuropathy sensitized with GD1b ganglioside, GD1b may be a target molecule for autoantibody in some patients with acute sensory ataxic neuropathy.[SDGs]SDG3immunoglobulin G antibody; acute disease; adult; antibody titer; article; ataxia; case report; disease association; female; human; nerve conduction; neuropathy; priority journal; sensory neuropathyjournal article10.1212/WNL.57.7.1316115918572-s2.0-0035833949