A genome-wide association analysis identifies novel susceptibility loci for coronary arterial lesions in patients with Kawasaki disease

MING-TAI LINCHIA-LIN HSUPEI-LUNG CHENWEI-SHIUNG YANGJOU-KOU WANGFann C.S.J.MEI-HWAN WU2020-12-212020-12-2120131931-5244https://www.scopus.com/inward/record.uri?eid=2-s2.0-84878111459&doi=10.1016%2fj.trsl.2013.02.002&partnerID=40&md5=5f9ca0b2c20c5fbf6b5d63406032dd53https://scholars.lib.ntu.edu.tw/handle/123456789/529305[SDGs]SDG3CD40 antigen; DNA fragment; platelet derived growth factor; Rac protein; Smad3 protein; transforming growth factor beta; article; coronary arterial lesion; coronary artery aneurysm; coronary artery disease; coronary risk; disease severity; female; gene frequency; gene locus; gene replication; genetic association; genetic susceptibility; genetic transcription; genetic variability; genotype; histopathology; human; intron; lymphocyte migration; lymphocytic infiltration; major clinical study; male; mucocutaneous lymph node syndrome; priority journal; risk factor; single nucleotide polymorphism; smooth muscle fiberA genome-wide association analysis identifies novel susceptibility loci for coronary arterial lesions in patients with Kawasaki diseasejournal article10.1016/j.trsl.2013.02.0022-s2.0-84878111459