MEI-HWEI CHANG2021-01-042021-01-0420020277-2116https://www.scopus.com/inward/record.uri?eid=2-s2.0-19044389450&doi=10.1097%2f00005176-200204000-00024&partnerID=40&md5=44374b45f5d71ec6954c8b0df9a106a9https://scholars.lib.ntu.edu.tw/handle/123456789/537132[SDGs]SDG3article; genetics; hepatitis B; Hepatitis B virus; human; infant; mutation; physiology; Hepatitis B; Hepatitis B virus; Humans; Infant; Mutation; MLCS; MLOWNViral mutants and fulminant hepatitis. A dominant hepatitis B virus population defective in virus secretion because of several S-gene mutations from a patient with fulminant hepatitis.journal article10.1097/00005176-200204000-00024119819522-s2.0-19044389450