Fu, Ssu-JuSsu-JuFuMENG-CHUN HUPeng, Yi-JhengYi-JhengPengFang, Hsin-YuHsin-YuFangHsiao, Cheng-TsungCheng-TsungHsiaoChen, Tsung-YuTsung-YuChenJeng, Chung-JiuanChung-JiuanJengCHIH-YUNG TANG2020-06-302020-06-3020202073-4409https://www.scopus.com/inward/record.uri?eid=2-s2.0-85085678879&doi=10.3390%2fcells9061332&partnerID=40&md5=13783715221c6fcf8fbc1c10b9977d34https://scholars.lib.ntu.edu.tw/handle/123456789/506979[SDGs]SDG3chloride channel; ClC-2 chloride channels; CRBN protein, human; Crbn protein, mouse; CUL4A protein, human; CUL4B protein, human; cullin; DDB1 protein, human; DNA binding protein; polyubiquitin; signal transducing adaptor protein; ubiquitin protein ligase; animal; biological model; brain disease; C57BL mouse; enzyme specificity; HEK293 cell line; human; hyperaldosteronism; metabolism; pathology; protein degradation; protein homeostasis; ubiquitination; Wistar rat; Adaptor Proteins, Signal Transducing; Animals; Brain Diseases; Chloride Channels; Cullin Proteins; DNA-Binding Proteins; HEK293 Cells; Humans; Hyperaldosteronism; Mice, Inbred C57BL; Models, Biological; Polyubiquitin; Proteolysis; Proteostasis; Rats, Wistar; Substrate Specificity; Ubiquitin-Protein Ligases; Ubiquitinationjournal article10.3390/cells9061332324664892-s2.0-85085678879