Chou C.-HSHEE-UAN CHENCHIN-DER CHENCHIA-TUNG SHUNWen W.-FYI-AN TUJEHN-HSIAHN YANG2020-02-132020-02-1320201945-7197https://scholars.lib.ntu.edu.tw/handle/123456789/458268[SDGs]SDG3acetylcysteine; adenosine triphosphate; chorionic gonadotropin; estradiol; estrogen receptor alpha; fulvestrant; manganese superoxide dismutase; mitochondrial DNA; reactive oxygen metabolite; ubidecarenone; estradiol; estrogen; animal cell; animal experiment; animal model; animal tissue; Article; cell function; clinical article; concentration (parameter); disorders of mitochondrial functions; electron microscopy; embryo; endometrium cell; endometrium polyp; female; human; human cell; human tissue; immunofluorescence; immunohistochemistry; in vitro study; in vivo study; mitochondrion; mouse; nidation; nonhuman; priority journal; animal; apoptosis; cell culture; drug effect; endometrium; epithelium cell; Institute for Cancer Research mouse; metabolism; pathology; Animals; Apoptosis; Cells, Cultured; Endometrium; Epithelial Cells; Estradiol; Estrogens; Female; Humans; Mice; Mice, Inbred ICR; Mitochondria; Reactive Oxygen SpeciesMitochondrial Dysfunction Induced by High Estradiol Concentrations in Endometrial Epithelial Cellsjournal article10.1210/clinem/dgz015315127262-s2.0-85076386474