LI-CHUN CHANGHAN-MO CHIUCHIA-TUNG SHUNJIN-TUNG LIANGLin, Jaw-TownJaw-TownLinCHIEN-CHUAN CHENYI-CHIA LEEMING-SHIANG WU2022-01-142022-01-142014-12-311471-230Xhttps://scholars.lib.ntu.edu.tw/handle/123456789/592397Investigations of genetic alterations and correlations with histology or morphology could provide further insights into colorectal carcinogenesis. Nevertheless, such genetic changes were less investigated in adenoma stage and a comprehensive survey of oncogenic mutations in EGFR signaling pathway according to different morphologic subtypes has not been performed.enGene mutation; Non-polypoid colorectal neoplasm; EGFR; LATERALLY-SPREADING TUMORS; K-RAS MUTATIONS; CHINESE POPULATION; SERRATED PATHWAY; COLON CANCERS; POOR SURVIVAL; STOOL DNA; FLAT; CARCINOMA; PREVALENCE[SDGs]SDG3B Raf kinase; epidermal growth factor receptor; epidermal growth factor receptor 2; protein kinase B; B Raf kinase; KRAS protein, human; oncoprotein; phosphatidylinositol 3 kinase; PIK3CA protein, human; Ras protein; adult; aged; AKT gene; Article; BRAF gene; cancer classification; cancer staging; carcinogenesis; colorectal adenoma; controlled study; disease severity; EGFR gene; epigenetics; exon; female; gene; genetic association; HER2 gene; histopathology; human; human tissue; immunohistochemistry; major clinical study; male; middle aged; morphology; mutational analysis; oncogene K ras; PIK3CA gene; tumor volume; adenoma; colorectal tumor; genetics; mutation; nucleotide sequence; pathology; Adenoma; Aged; Colorectal Neoplasms; DNA Mutational Analysis; Female; Humans; Male; Middle Aged; Mutation; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins B-raf; ras Proteinsjournal article10.1186/s12876-014-0221-y255516252-s2.0-84965084561WOS:000347984300001https://scholars.lib.ntu.edu.tw/handle/123456789/544688