Wu, Sheng-MaoSheng-MaoWuJan, Yee-JeeYee-JeeJanTsai, Shih-ChuanShih-ChuanTsaiPan, Hung-ChuanHung-ChuanPanShen, Chin-ChangChin-ChangShenYang, Cheng-NingCheng-NingYangLee, Shu-HuaShu-HuaLeeSHING-HWA LIUShen, Li-WeiLi-WeiShenChiu, Chien-ShanChien-ShanChiuArbiser, Jack LJack LArbiserMeng, MenghsiaoMenghsiaoMengSheu, Meei-LingMeei-LingSheu2023-02-212023-02-212022-01-010742-2091https://scholars.lib.ntu.edu.tw/handle/123456789/628581Histone deacetylase (HDAC) inhibitors (HDIs) can modulate the epithelial-mesenchymal transition (EMT) progression and inhibit the migration and invasion of cancer cells. Emerging as a novel class of anti-cancer drugs, HDIs are attracted much attention in the field of drug discovery. This study aimed to discern the underlying mechanisms of Honokiol in preventing the metastatic dissemination of gastric cancer cells by inhibiting HDAC3 activity/expression.enEndoplasmic reticulum stress; Epithelial–mesenchymal transition; Histone deacetylase 3; Histone deacetylase inhibitors; Peritoneal dissemination[SDGs]SDG3journal article10.1007/s10565-021-09673-2349731352-s2.0-85122003611WOS:000736942000001https://api.elsevier.com/content/abstract/scopus_id/85122003611