Chen, Chien-HanChien-HanChenYUNG-LING LEEWu, Ming-HsunMing-HsunWuChen, Pao-JenPao-JenChenWei, Tien-ShanTien-ShanWeiPAU-CHUNG CHENTseng , Ching-IngChing-IngTsengWEI J. CHEN2020-02-052020-02-052019-080954-7894https://scholars.lib.ntu.edu.tw/handle/123456789/453472Elevated cord blood IgE (cIgE), a predictor of atopic diseases, is influenced by genetic and environmental factors. However, gene-environment interactions on cIgE elevation and their difference by sex remain largely unexplored.enIgE; cord blood; prenatal environment; sex; single nucleotide polymorphism[SDGs]SDG3DNA; immunoglobulin E; interleukin 13; interleukin 4; STAT6 protein; tobacco smoke; water; IL4 protein, human; immunoglobulin E; interleukin 13; interleukin 4; STAT6 protein; STAT6 protein, human; adult; Article; cohort analysis; controlled study; female; genetic variability; genotype; genotype environment interaction; household; human; immunoglobulin blood level; major clinical study; male; mold; newborn; odor; prenatal exposure; priority journal; prospective study; sex; single nucleotide polymorphism; structured questionnaire; third trimester pregnancy; umbilical cord blood; fetus blood; genetics; hypersensitivity; immunology; passive smoking; pregnancy; questionnaire; sexual characteristics; single nucleotide polymorphism; Female; Fetal Blood; Humans; Hypersensitivity; Immunoglobulin E; Infant, Newborn; Interleukin-13; Interleukin-4; Male; Polymorphism, Single Nucleotide; Pregnancy; Prenatal Exposure Delayed Effects; Sex Characteristics; STAT6 Transcription Factor; Surveys and Questionnaires; Tobacco Smoke Pollutionjournal article10.1111/cea.13419311024812-s2.0-85070227865WOS:000478767800008https://www.scopus.com/inward/record.uri?eid=2-s2.0-85070227865&doi=10.1111%2fcea.13419&partnerID=40&md5=c5027232171b8452cddff37d44e840a4