Effect of Hyperoxia on Post-Resuscitation Neurological Injury: Exploring Cerebral Hemodynamics, Tissue Perfusion, Blood-Brain-Barrier Disruption, Akt-eNOS Mediated NO Release in Endothelium and Anti-Apoptotic Protection and Mitochondrial Function In=高血氧對心肺復甦後的神經學傷害---從腦部血行動力學、組織灌流及血腦屏障破壞出發，探討高血氧對血管內皮Akt-eNOS途徑導引一氧化氮生成、血管擴張、以及對神經細胞本身抗凋亡保護與粒線體功能的影響

2014-08-012024-05-13https://scholars.lib.ntu.edu.tw/handle/123456789/646376高氧氧氣缺血再灌流反應性氧化物神經內皮細胞微循環粒線體呼吸鏈細胞凋亡hyperoxiaoxygenischemiareperfusion injuryreactive oxygen speciesneuronendotheliummicrocirculationmitochondriarespiratory chainapoptosisEffect of Hyperoxia on Post-Resuscitation Neurological Injury: Exploring Cerebral Hemodynamics, Tissue Perfusion, Blood-Brain-Barrier Disruption, Akt-eNOS Mediated NO Release in Endothelium and Anti-Apoptotic Protection and Mitochondrial Function In=高血氧對心肺復甦後的神經學傷害---從腦部血行動力學、組織灌流及血腦屏障破壞出發，探討高血氧對血管內皮Akt-eNOS途徑導引一氧化氮生成、血管擴張、以及對神經細胞本身抗凋亡保護與粒線體功能的影響