WANG-TSO LEELi, Yue-JuYue-JuLiWu, Shi-BeiShi-BeiWuYang, Cheng-NingCheng-NingYangWu, Tai-ShengTai-ShengWuWei, Yau-HueiYau-HueiWeiDeng, Yi-TingYi-TingDeng2019-11-192019-11-192018-030929-6646https://scholars.lib.ntu.edu.tw/handle/123456789/431452Deregulation of metabolic pathways is one of the hallmarks of cancer progression. Connective tissue growth factor (CTGF/CCN2) acts as a tumor suppressor in oral squamous cell carcinoma (OSCC). However, the role of CTGF in modulating cancer metabolism is still unclear.enCTGF; Metabolism; Mitochondrion; OSCC; mtTFA[SDGs]SDG3[SDGs]SDG14adenosine triphosphate; benzyloxycarbonylleucylleucylleucinal; connective tissue growth factor; mitochondrial DNA; mitochondrial transcription factor A; short hairpin RNA; ubiquitin; ubiquitinated protein; connective tissue growth factor; DNA binding protein; mitochondrial protein; mitochondrial transcription factor A; transcription factor; ubiquitin; acidification; Article; cell invasion; cell migration; copy number variation; glycolysis; immunoprecipitation; mitochondrial biogenesis; mitochondrial respiration; mouth squamous cell carcinoma; oxygen consumption; protein expression; real time polymerase chain reaction; SAS cell line; Western blotting; human; metabolism; mitochondrion; mouth tumor; physiology; squamous cell carcinoma; tumor cell line; Carcinoma, Squamous Cell; Cell Line, Tumor; Connective Tissue Growth Factor; DNA-Binding Proteins; Humans; Mitochondria; Mitochondrial Proteins; Mouth Neoplasms; Transcription Factors; Ubiquitinjournal article10.1016/j.jfma.2017.04.003284384342-s2.0-85018732180WOS:000426952600008https://api.elsevier.com/content/abstract/scopus_id/85018732180