Repository logo
  • English
  • 中文
Log In
Have you forgotten your password?
  1. Home
  2. College of Life Science / 生命科學院
  3. Life Science / 生命科學系
  4. Arginine vasopressin modulates ion and acid/base balance by regulating cell numbers of sodium chloride cotransporter and h+-atpase rich ionocytes
 
  • Details

Arginine vasopressin modulates ion and acid/base balance by regulating cell numbers of sodium chloride cotransporter and h+-atpase rich ionocytes

Journal
International Journal of Molecular Sciences
Journal Volume
21
Journal Issue
11
Pages
3957
Date Issued
2020
Author(s)
Tong S.-K
Lee H.-L
Lee Y.-C
Wu L.-C
Tsou Y.-L
Lu S.-W
Shih S.-W
Hwang P.-P
Chou M.-Y.
MING-YI CHOU  
DOI
10.3390/ijms21113957
URI
https://www.scopus.com/inward/record.uri?eid=2-s2.0-85085757592&doi=10.3390%2fijms21113957&partnerID=40&md5=cca02644bd02078ecff6d502d862ec8f
https://scholars.lib.ntu.edu.tw/handle/123456789/581545
Abstract
Arginine vasopressin (Avp) is a conserved pleiotropic hormone that is known to regulate both water reabsorption and ion balance; however, many of the mechanisms underlying its effects remain unclear. Here, we used zebrafish embryos to investigate how Avp modulates ion and acid– base homeostasis. After incubating embryos in double-deionized water for 24 h, avp mRNA expression levels were significantly upregulated. Knockdown of Avp protein expression by an antisense morpholino oligonucleotide (MO) reduced the expression of ionocyte-related genes and downregulated whole-body Cl- content and H+ secretion, while Na+ and Ca2+ levels were not affected. Incubation of Avp antagonist SR49059 also downregulated the mRNA expression of sodium chloride cotransporter 2b (ncc2b), which is a transporter responsible for Cl- uptake. Correspondingly, avp morphants showed lower NCC and H+-ATPase rich (HR) cell numbers, but Na+/K+-ATPase rich (NaR) cell numbers remained unchanged. avp MO also downregulated the numbers of foxi3a-and p63-expressing cells. Finally, the mRNA expression levels of calcitonin gene-related peptide (cgrp) and its receptor, calcitonin receptor-like 1 (crlr1), were downregulated in avp morphants, suggesting that Avp might affect Cgrp and Crlr1 for modulating Cl- balance. Together, our results reveal a molecular/cellular pathway through which Avp regulates ion and acid–base balance, providing new insights into its function. ? 2020 by the authors. Licensee MDPI, Basel, Switzerland.
Subjects
adenosine triphosphatase (potassium sodium); antisense oligonucleotide; argipressin; calcitonin gene related peptide; calcitonin gene related peptide receptor; calcitonin receptor like receptor; cystic fibrosis transmembrane conductance regulator; isotocin; messenger RNA; nelivaptan; prolactin; protein p63; proton transporting adenosine triphosphatase; relcovaptan; sodium chloride cotransporter; sodium ion; tolvaptan; antisense oligonucleotide; argipressin; calcitonin gene related peptide; calcium; chloride; complementary DNA; ion; messenger RNA; sodium; sodium chloride cotransporter; vasopressin derivative; zebrafish protein; acid base balance; animal tissue; Article; brain tissue; cell count; controlled study; down regulation; embryo; epithelium cell; gene knockdown; gill; heart tissue; ion transport; ionocyte; kidney tissue; mRNA expression level; muscle tissue; nonhuman; osmoregulation; protein expression; salinity; zebra fish; animal; chemistry; electrode; genetics; homeostasis; in situ hybridization; ion transport; metabolism; skin; Acid-Base Equilibrium; Animals; Arginine Vasopressin; Calcitonin Gene-Related Peptide; Calcium; Chlorides; DNA, Complementary; Down-Regulation; Electrodes; Homeostasis; In Situ Hybridization; Ion Transport; Ions; Oligonucleotides, Antisense; RNA, Messenger; Skin; Sodium; Sodium Chloride Symporters; Vasopressins; Zebrafish; Zebrafish Proteins
Type
journal article

臺大位居世界頂尖大學之列,為永久珍藏及向國際展現本校豐碩的研究成果及學術能量,圖書館整合機構典藏(NTUR)與學術庫(AH)不同功能平台,成為臺大學術典藏NTU scholars。期能整合研究能量、促進交流合作、保存學術產出、推廣研究成果。

To permanently archive and promote researcher profiles and scholarly works, Library integrates the services of “NTU Repository” with “Academic Hub” to form NTU Scholars.

總館學科館員 (Main Library)
醫學圖書館學科館員 (Medical Library)
社會科學院辜振甫紀念圖書館學科館員 (Social Sciences Library)

開放取用是從使用者角度提升資訊取用性的社會運動,應用在學術研究上是透過將研究著作公開供使用者自由取閱,以促進學術傳播及因應期刊訂購費用逐年攀升。同時可加速研究發展、提升研究影響力,NTU Scholars即為本校的開放取用典藏(OA Archive)平台。(點選深入了解OA)

  • 請確認所上傳的全文是原創的內容,若該文件包含部分內容的版權非匯入者所有,或由第三方贊助與合作完成,請確認該版權所有者及第三方同意提供此授權。
    Please represent that the submission is your original work, and that you have the right to grant the rights to upload.
  • 若欲上傳已出版的全文電子檔,可使用Open policy finder網站查詢,以確認出版單位之版權政策。
    Please use Open policy finder to find a summary of permissions that are normally given as part of each publisher's copyright transfer agreement.
  • 網站簡介 (Quickstart Guide)
  • 使用手冊 (Instruction Manual)
  • 線上預約服務 (Booking Service)
  • 方案一:臺灣大學計算機中心帳號登入
    (With C&INC Email Account)
  • 方案二:ORCID帳號登入 (With ORCID)
  • 方案一:定期更新ORCID者,以ID匯入 (Search for identifier (ORCID))
  • 方案二:自行建檔 (Default mode Submission)
  • 方案三:學科館員協助匯入 (Email worklist to subject librarians)

Built with DSpace-CRIS software - Extension maintained and optimized by 4Science