Mitochondrial Na+ overload is caused by oxidative stress and leads to activation of the caspase 3-dependent apoptotic machinery
Journal
FASEB Journal
Journal Volume
18
Journal Issue
12
Pages
1442-1444
Date Issued
2004
Author(s)
SDGs
Other Subjects
caspase 3; hydrogen peroxide; sodium calcium exchange protein; sodium ion; animal cell; apoptosis; article; calcium cell level; cardiovascular disease; controlled study; heart muscle cell; membrane potential; mitochondrial membrane; mitochondrion; nonhuman; oxidative stress; priority journal; signal transduction; sodium cell level; Apoptosis; Calcium; Caspase 3; Caspases; Cytochromes c; Cytosol; Enzyme Activation; Hydrogen Peroxide; Mitochondria; Myocytes, Cardiac; Oxidative Stress; Signal Transduction; Sodium; Sodium-Calcium Exchanger; Animalia
Type
journal article